Background: The hippocampus is a specialized brain region that governs learning, memory and mood. During ageing, the hippocampus shrinks and this is hypothesized to contribute to age-related cognitive decline. Within this PhD project, the student will use a combination of statistical genetic approaches and cellular neuroscience to explore the mechanisms underlying age-related hippocampal volume loss. They will further explore whether specific blood-based factors are capable of modifying the rate of volume loss by stimulating adult hippocampal neurogenesis.
Overarching aim: To equip an enthusiastic PhD student with a multidisciplinary skill set that they will use to explore why our hippocampi shrink with age and how we might preserve hippocampal volume for longer, in order to slow down cognitive decline.
Techniques: Transcriptome-wide association studies; Mendelian Randomisation; Neural Stem Cell Culture; Genome editing (e.g., CRISPR) or gene overexpression and downregulation; gene expression analyses (e.g., scRNA Seq, qPCR), immunocytochemistry and high-content imaging.
Objectives for each year:
Year 1 (Rotation): To use neuroimaging genome-wide association study data to perform a transcriptome-wide association study in order to impute which genes are upregulated or downregulated in the hippocampus in association with greater age-related volume loss.
Year 2: To test if genetic risk factors for age-related hippocampal loss exert their effects by impacting adult hippocampal neurogenesis, using an in vitro model.
Year 3: To infer whether specific blood-based factors modify rates of age-related hippocampal loss and cognitive decline, using large genetic datasets and Mendelian Randomisation.
Year 4: To test whether specific blood-based factors protect hippocampal cells from premature cell ageing, using an in vitro model.